Celva / Treatments / Neurologic, cardiovascular & autoimmune

Medically reviewed by the Celva medical team · June 2026

§ 003 / Treatment · Evaluation-first

We'll tell you
honestly if this
can help.

This is our work with neurologic, cardiovascular, and autoimmune disease: the conditions standard care often can't reach. For the right patient, at the right stage, cell therapy can be worth trying. We run a careful evaluation first, and if the honest answer is "not this therapy," we'll say so.

Review
3MDs
physicians review every case before treatment
Dose
Setper case
Weight-adjusted, built for your case, never off the shelf
Baseline
12+
Functional measures captured before dose one
Two Celva clinicians reviewing brain MRI slices on a monitor during a multi-disciplinary case review
FIG. 03 · Multi-disciplinary review Specialty + regenerative, together
§ 003.1 / What we tell you first

Cell therapy for complex disease
is real, partial, and
still being understood.

There is credible evidence that mesenchymal stem cells can modulate neuroinflammation and, in specific conditions, support function. There is not yet credible evidence that they reverse established neurodegeneration, regrow myelin at scale, or restore lost neurons in a clinically meaningful way.

We operate in the window between those two statements. For some patients (with the right condition, the right disease stage, and realistic expectations) that window is worth entering. For many, it isn't.

And it is rarely one treatment. The response tends to build as treatments stack, and progressive conditions usually need ongoing care to hold ground and extend it. A process, not a cure.

Everything below is written assuming you want the real picture, not the marketing one.

§ 003.2 / Candidacy

Could this help you?
Let's look.

Cell therapy doesn't work by diagnosis. It works on biology: the inflammation, immune signaling, and tissue your body still has to work with. So the real question isn't whether your exact diagnosis sits on a list. It's whether your situation has something we can influence, and that's usually only clear once we look.

What we look at · 01

Active biology

Is there inflammation or immune activity to influence? That signaling environment is where MSCs do most of their work, and it shows up across very different diagnoses.

What we look at · 02

Tissue to work with

Is there still structure to support? Earlier in a process usually gives us more to build on than later, which is a reason to ask sooner rather than wait it out.

What we look at · 03

Stable footing

Are you steady on your current care, with your own specialists still directing it? We add to that. We don't replace your medication or your doctors.

What we look at · 04

Realistic goals

We aim for meaningful change, not miracles. Knowing what "better" would mean for you is part of deciding whether this is worth doing at all.

Where this comes up most

Neurologic, cardiovascular, autoimmune, and post-viral conditions: from Parkinson's and post-stroke recovery to MS, dysautonomia, and ischemic heart disease. These are examples, not a checklist.

When cell therapy isn't the right tool, like advanced structural loss or an urgent problem that needs a hospital now, we'll say so and point you somewhere better. We would rather turn you away honestly than sell hope we can't support.

Get an honest read on your case →
§ 003.25 / Mechanism · systemic

How the cells
work systemically.

For systemic and complex indications the goal is to recalibrate the upstream biology (chronic inflammation, dysregulated immune activity, compromised perfusion, neuroinflammation where present), so the body's own repair machinery can function again. The signaling persists well beyond the cells themselves.

How the cells work: six simultaneous mechanisms

Anti-inflammatory

Reduces the chronic inflammatory signaling that contributes to pain, dysfunction, and progressive tissue damage, systemically.

Anti-fibrotic

Attenuates the fibrotic cascade that replaces functional tissue with scar tissue, a downstream consequence of chronic inflammation.

Immunomodulatory

Recalibrates dysregulated immune activity across the body without systemic immune suppression.

Pro-angiogenic

Supports formation of new vascular networks, contributing to tissue perfusion and a more favorable systemic repair environment.

Neurotrophic

Releases growth factors (BDNF, NGF, GDNF) that support neuronal survival and protect nerve tissue from chronic inflammatory damage.

Anti-apoptotic

Signals stressed, at-risk host cells to survive rather than die, mediated by factors like HGF and IGF-1, preserving tissue that chronic inflammation would otherwise lose.

Why immunomodulation leads

Systemic and complex conditions, autoimmune, neurologic, post-viral, cardiovascular inflammation, often have immune dysregulation at their core. MSCs work because they recalibrate the immune environment without globally suppressing it the way steroids and biologics do. The downstream effects on inflammation, fibrosis, perfusion, and neurotrophic support follow from the same primary mechanism.

Why we match the cell to the case

We don't use one cell off the shelf. For neurologic cases, we build on umbilical-cord (Wharton's jelly) MSCs specifically because they secrete higher levels of neurotrophic factors, BDNF, NGF, and GDNF, than marrow-derived cells. That richer signaling supports neuronal survival and the nerve's own repair environment. It's signaling, not tissue replacement, which is also why it has honest limits.

§ 003.3 / Evaluation

The whole team weighs in,
before any decision.

Before anyone is approved, we gather your complete history and imaging, and the medical team reviews and rounds on your case together. From that, they build a treatment plan made for you, or give you an honest answer that it isn't the right path. We take the time to get it right, not to drag it out.

  1. 01

    Intake & records

    Full medical history, imaging, specialist notes, prior treatment trials. We need everything, including what didn't work.

  2. 02

    Coordinator intake & physician team review

    45-minute call with a Celva patient coordinator. Celva's medical team then reviews independently, focused on disease stage, medication stability, and whether the evaluation should proceed. Not a sales call.

  3. 03

    Multi-disciplinary review

    Each case is reviewed by the physician team's regenerative attending with outside specialty consultation (neurology, rheumatology, cardiology, or other as relevant) and, where appropriate, PM&R review. Multiple opinions inform every decision.

  4. 04

    Baseline capture

    If we're considering acceptance: functional testing, autonomic battery, disease-specific scales (EDSS, COMPASS-31, Fugl-Meyer, etc.). Captured before any decision is finalized.

  5. 05

    Decision & plan

    One of three outcomes, in writing:
    Accept, with a specific protocol, expected magnitude, and measurement plan.
    Decline, with the reason, and any referrals we can make.
    Revisit, with the conditions under which we would re-open the case.

How selective we are

Complex-disease cohort

We are selective in patient acceptance and only move forward when a case appears clinically appropriate and expectations are realistic. Each application moves through intake, records screen, multi-disciplinary panel review, and a written plan. Cases that don't fit are declined in writing, with a referral when one is available.

§ 003.4 / Protocol (if accepted)

What treatment
looks like, for accepted patients.

Systemic and complex protocols are tailored, and no two patients get the same recipe. Most use umbilical-cord MSCs by IV as the foundation, often blended with bone-marrow MSCs depending on your indication, disease stage, and history. For neurologic cases the cord (Wharton's jelly) cells are chosen for their stronger neurotrophic secretome, which supports neuronal survival. Dose is weight-adjusted and tuned to severity. Route and cadence depend on your case; the common architecture is below, and the specifics live in your written plan. Why we use multiple cell types →

Source
Multiple allogeneic cell types, matched to your case
Dose
Weight-adjusted, set per case
Delivery
IV
Sessions
2–3 per year
Re-evaluation
At each check-in
Your records
Provided to you in full
Exit criteria
Agreed with you up front
01 / First treatment

First treatment & observation

Your first infusion, given under our attending physician's supervision, with structured follow-up in the weeks after.

Same day on site · post-dose labs included
02 / First review

First decision point

Around three months, we review your progress together: how you feel, your symptoms, and what the labs show. We weigh the full picture, not one number. A further dose is never automatic.

Reviewed together · continue / pause / stop
03 / If you're responding

We build on it

When you're responding, treatment continues, with dose and timing adjusted to how you respond. These conditions usually take more than one round, and the benefit tends to grow as doses stack. We keep reviewing at every visit.

Conditional on each review
04 / When we wrap up

Consolidation & exit

When the benefit has consolidated, or further treatment isn't adding enough, we bring the program to a close. You leave with your full records and a clear picture of what changed and what to keep an eye on.

Records provided to you
Rarely one and done

For complex and progressive disease, the response usually builds as treatments stack, each one layering on the last, and that's where the most durable benefit comes. It is not a cure: with a progressive condition, the work is to hold ground and keep extending it, which takes ongoing treatment. We continue only when it's clearly helping.

§ 003.5 / A case we declined
"I wanted so badly to accept him. He'd already flown to four clinics. What he needed, what would have been honest, was hospice planning and time with his family. We said so. He thanked us. That's the one I think about."
Dr. Alejandro Castillo, MD
Regenerative medicine attending · physician team

We turn people away when it is the honest thing to do, and we tell them why. That is the standard every case here is held to: we do not treat people we cannot truly help, and the most common reason we say no is simply that the disease is too far along for this to make a difference.

§ 003.6 / Dig deeper

Track-specific
reading.

For patients and families who want the detailed read on candidacy, expectations, and how the evaluation actually works. One page per topic.

§ 003.7 / Questions

Track-specific
questions.

Q.01 Why are you so selective about who you accept?
Because the biology here is harder (neuroinflammation, autoimmunity, post-viral states, vascular disease), and the harm of selling false hope is measured in a family's time, energy, and trust. Even among carefully chosen patients, not everyone responds. Accepting everyone who wanted treatment would erode the honesty of our outcomes, and the trust patients place in our accepting them.
Q.02 Do you coordinate with my specialist?
We're glad to involve them. In practice, many U.S. specialists can't formally recommend or coordinate on a therapy outside standard care, so we don't require your specialist's sign-off to move forward. What we do is give you your full records to share with whomever you choose. If your specialist is open to it, we'll share directly; if not, you leave with everything they would need to pick your care back up.
Q.03 What happens if nothing measurable changes in my first 90 days?
A flat reading at three months is common, and on its own it isn't a reason to stop. These are complex conditions that usually take more than one treatment, and the response tends to build as treatments stack, so in most cases a second treatment is reasonable. We review the full picture with you, how you feel and function alongside any labs, and continue while there is a real reason to. If it genuinely isn't helping, we say so and stop, with a written summary of what we measured, expected, and observed. We won't keep dosing without a reason.
Q.04 Is this a clinical trial?
No. This is a physician-led treatment program licensed in Mexico and regulated by COFEPRIS. We measure outcomes rigorously and publish them, but we are not an IRB-registered trial site. If a trial exists for your condition at a major center and you might qualify, we'll tell you. Sometimes that's the honest recommendation.
Q.05 What's the downside I should know about?
Two. First: there is a real chance that the treatment does not produce measurable change, and the program may end without the result you hoped for. Second: unlike joint patients, systemic and complex patients often face the slow, ambiguous grief of "did it work?" We structure measurement to cut through that. Not every patient wants that level of cold clarity, and it matters to know that going in.
§ 003.8 · Apply

Start with
the evaluation.

Not a sales call. A records review, a screen, and a multi-disciplinary opinion. If we accept you, we'll tell you what to expect. In writing. If we don't, we'll tell you why, and where we think you should look next.

See if you're a candidate →
These therapies are not FDA-approved. Treatments are performed in Mexico by the physician team at Hospital Angeles, Tijuana, regulated by COFEPRIS. Individual results may vary. Not medical advice. All patients undergo rigorous screening before treatment is recommended.